Currach on the shore in [[Inishbofin, Galway]]

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a.
FADD: Essential for Embryo Development and Signaling from Some, but Not All, Inducers of Apoptosis
Molecular basis of myotonic dystrophy: expansion of a trinucleotide (CTG) repeat at the 3' end of a transcript encoding a protein ki...
E1A signaling to p53 involves the p19(ARF) tumor suppressor.
Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes.
bax-Deficiency Promotes Drug Resistance and Oncogenic Transformation by Attenuating p53-Dependent Apoptosis
INK4a/ARF mutations accelerate lymphomagenesis and promote chemoresistance by disabling p53.
Expansion of a CUG trinucleotide repeat in the 3' untranslated region of myotonic dystrophy protein kinase transcripts results in nu...
Direct coupling of the cell cycle and cell death machinery by E2F.
Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear鈥塩hanges